Apparently, people suffering from diabetes encounter a significantly higher peril of osteoporotic fractures compared to those without the disease, but the rationale for this is not clear and cannot be satisfactorily predicted. Scientists from RPI (Rensselaer Polytechnic Institute) will utilize new measurement methods to observe—on an atomic level—the consequences type I diabetes has on bone density and how medication can reduce the perils of fracture. Deepak Vashishth—Director of the CBIS (Center for Biotechnology and Interdisciplinary Studies)—said, “We are looking at the systems of what can lead to such a fragility.”
The research was being done in collaboration with CU (Creighton University) and is being backed by a new R01 grant from the NIH’s NIDDK (National Institute of Diabetes and Digestive and Kidney Diseases). Vashishth said this kind of understanding is important as fractures cannot be completely explained or estimated by using existing clinical tools. He added people having type I diabetes, experience a higher lifetime peril of fracture compared to those having type II diabetes.
On a related note, recently, a study showed that family medical history of diabetes is associated with augmented bone mineral density. The link amid type II diabetes and surged fracture peril is well documented. Nonetheless, the possible impact of family history of diabetes on the BMD (bone mineral density) is less known. Research from China now substantiates that a history of first-degree family members having diabetes is interrelated to amplified BMD plus to insulin resistance. The study findings were published in Menopause, which is the journal of the NAMS (North American Menopause Society). As patients having type II diabetes are at greater risk of fracture, knowing the early pathophysiology of changed BMD can be critical in the advancement of preventive tactics for diabetic osteoporosis.
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